SENS Foundation is Hiring
The SENS Foundation, freshly infused with money from a recent half-million dollar donation, is hiring a new research assistant for their California research center. As you'll all know by now, the Foundation works on developing rejuvenation biotechnologies to repair and reverse the damage of aging. Do you want to be a part of that grand and noble effort? Then read on.
SENS Foundation seeking to hire Research Assistant
SENS Foundation is hiring a Research Assistant for our research center located in Mountain View, CA. Qualified candidates will a BS or MS in the chemical/biological sciences and two years of work experience. Experience should be in using standard equipment, including but not limited to standard bench cell biology/biochem/molecular biology techniques. Good fundamental laboratory skills to include safety, excellent pipetting skill, and mammalian cell culture.Duties will include cell culture, transfection, microscopy, and protein work (western blot, IP, etc.). Subcloning / PCR cloning a plus. Experience working with mitochondria a strong plus. Research assistants will work under the supervision of the facility's senior researcher to carry out experiments directed towards establishing the viability of the SENS mission and chosen therapeutic goals. Research Assistants will receive general instruction on routine work and detailed instruction on new assignments. Other duties include providing support in areas such as lab maintenance, maintaining lab notes, procuring supplies, organizing research materials, and editing and filing documentation.
They are looking for mitochondrial research experience because the MitoSENS project is forging ahead. The goal of this line of work is to duplicate vulnerable mitochondrial genes in the cell nucleus, while enabling the vital proteins they produce to make their way back to the mitochondria. Thus even if the genes become damaged in the mitochondria, there will be a backup source of proteins to avoid the normal consequences of mitochondrial DNA damage.
For over 30 years mutations in mitochondrial DNA have been suspected to be important contributors to aging. If we can incorporate working copies of that mtDNA into our nuclear DNA, the mtDNA will be rendered superfluous and any mutations it suffers will be inconsequential.
Do you have laboratory experience? Then apply. After all, there is no more important task than helping to eliminate degenerative aging and age-related disease. No other area of present human endeavor has the potential to save as many lives and relieve as much suffering.